"At the systems neuroscience level, ketamine’s mechanisms are markedly distinct from drugs such as propofol or the halogenated ethers. Unlike most anesthetics, ketamine does not appear to activate the sleep-promoting ventrolateral preoptic nucleus of the hypothalamus; rather, ketamine activates subcortical wake-promoting nuclei (Lu et al., 2008). This is paralleled in ketamine’s unique neurochemistry. Unlike most other inhaled and IV anesthetics, ketamine increases levels of the
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